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The invention relates to an apparatus for identifying the spatial location of a pattern recorded on a medium on the basis of a threshold value.
b) Description of the Prior Art
At present, documents, for example, preprinted business forms, are scanned one after another using a document reader, such as a scanner. The document reader is connected to a computer. This procedure is employed for storing the documents or for digitizing them. When, however, the documents are successively checked, it is possible that one of the documents being checked has already been read and stored in the computer. In this case, the computer still performs readout operations, during the interval before the next document is read in, which operations lead to a disproportionately high computing outlay., the aetiology of peripheral dysfunction due to acetaminophen toxicity is due to lack of myoendothelial cell injury \[[@CR15]\]. Furthermore, the primary function of the small intestinal mucosal cells is to maintain the intestinal barrier \[[@CR8]\]. Any disruption to this barrier induces an inflammatory response, and hence, this could be a plausible explanation as to why the small intestine is the first site of acetaminophen toxicity. However, several authors have found that co-administration of pre-existing acetaminophen with regional anaesthesia increased the risk of mucosal sloughing and serosal necrosis compared with the use of these anaesthetics alone \[[@CR17]–[@CR20]\]. Thus, it has been suggested that regional anaesthesia by itself may promote gut dysfunction.

We found the risk of requiring NMBD to be increased with the concomitant administration of opioids and acetaminophen. The mechanism of action of NMBD in maintaining post-operative muscle tone is by increased synthesis of myosin heavy chain and muscle contractility \[[@CR21]\]. Opioids inhibit myosin light chain kinase activity and contractility, which may have an inhibitory effect on NMBD activity and hence lower muscle tone \[[@CR21]–[@CR23]\]. It is possible that this may result in an increased requirement for NMBD due to impaired relaxation of the intestinal smooth muscle. Animal studies have shown that acute NMBD is able to normalise the rate
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